Because of a lapse in government funding, the information on this
website may not be up to date, transactions submitted via the
website may not be processed, and the agency may not be able to
respond to inquiries until appropriations are enacted. The NIH
Clinical Center (the research hospital of NIH) is open. For more
details about its operating status, please visit
cc.nih.gov. Updates
regarding government operating status and resumption of normal
operations can be found at
opm.gov.
Parkinson Disease, Postencephalitic MeSH Descriptor Data 2026
Parkinsonism following encephalitis, historically seen as a sequella of encephalitis lethargica (Von Economo Encephalitis). The early age of onset, the rapid progression of symptoms followed by stabilization, and the presence of a variety of other neurological disorders (e.g., sociopathic behavior; TICS; MUSCLE SPASMS; oculogyric crises; hyperphagia; and bizarre movements) distinguish this condition from primary PARKINSON DISEASE. Pathologic features include neuronal loss and gliosis concentrated in the MESENCEPHALON; SUBTHALAMUS; and HYPOTHALAMUS. (From Adams et al., Principles of Neurology, 6th ed, p754)
Entry Version
PARKINSON DIS POSTENCEPH
Entry Term(s)
Encephalitis Lethargica Type Parkinsonism
Parkinson Disease, Post-Encephalitic
Parkinsonian Syndrome, Postencephalitis
Parkinsonism, Postencephalitic
Parkinsonism, Viral Meningoencephalitic
Post-Encephalitic Parkinson Disease
Postencephalitic Economo-Type Parkinsonism
Postencephalitic Parkinson Disease
Postencephalitic Parkinsonism
Viral Meningoencephalitic Parkinsonism
von Economo Encephalitis Type Parkinsonism
Previous Indexing
Parkinson Disease (1966)
Public MeSH Note
1979; see PARKINSONISM, POSTENCEPHALITIC 1967-1978
Parkinsonism following encephalitis, historically seen as a sequella of encephalitis lethargica (Von Economo Encephalitis). The early age of onset, the rapid progression of symptoms followed by stabilization, and the presence of a variety of other neurological disorders (e.g., sociopathic behavior; TICS; MUSCLE SPASMS; oculogyric crises; hyperphagia; and bizarre movements) distinguish this condition from primary PARKINSON DISEASE. Pathologic features include neuronal loss and gliosis concentrated in the MESENCEPHALON; SUBTHALAMUS; and HYPOTHALAMUS. (From Adams et al., Principles of Neurology, 6th ed, p754)